The growth of new blood vessels from existing ones — essential for wound healing, but also the process tumors exploit to feed themselves.BPC-157 and GHK-Cu have documented pro-angiogenic effects via VEGFR2 upregulation and VEGF stimulation respectively. Angiogenesis is therapeutically desirable in wound healing and ischemic tissue but pathological in tumor growth and retinal neovascularization. The same compound that promotes healing-appropriate angiogenesis in one context could theoretically worsen another condition — context and tissue matter.